Centre for Experimental Cardiovascular Research Institute of Physiology Academy of Sciences of the Czech Republic Prague Czech Republic

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Murray CJ, Lopez AD. Alternative projections of mortality and disability by cause 1990–2020: Global Burden of Disease Study. Lancet. 1997;349:1498–504. PubMed

Braunwald E. The Simon Dack lecture. Cardiology: The past, the present, and the future. J Am Coll Cardiol. 2003;42:2031–41. PubMed

Stanek V. [Progress in the therapy of ischemic heart disease.] Kapitoly z kardiologie. 2002;4:3–11.

Julian DG. Treatment of cardiac arrest in acute myocardial ischaemia and infarction. Lancet. 1961;ii:840–4. PubMed

Mehta NJ, Khan IA. Cardiology’s 10 greatest discoveries of the 20th century. Tex Heart Inst J. 2002;29:164–71. PubMed PMC

Hurtado A. Some clinical aspects of life at high altitudes. Ann Intern Med. 1960;53:247–58. PubMed

Oštádal B, Oštádalová I, Kolár F, Pelouch V, Dhalla NS. Cardiac adaptation to chronic hypoxia. Adv Organ Biol. 1998;6:43–60.

Maroko PR, Kjekshus JK, Sobel BE, et al. Factors influencing infarct size following experimental coronary artery occlusion. Circulation. 1971;43:67–82. PubMed

Murry CE, Jennings RB, Reimer KA. Preconditioning with ischemia: A delay of lethal cell injury in ischemic myocardium. Circulation. 1986;74:1124–36. PubMed

Marber MS, Latchman DS, Walker JM, Yellon DM. Cardiac stress protein elevation 24 h after brief ischemia or heat stress is associated with resistance to myocardial infarction. Circulation. 1993;88:1264–72. PubMed

Yellon DM, Downey JM. Preconditioning the myocardium: From cellular physiology to clinical cardiology. Physiol Rev. 2003;83:1113–51. PubMed

Kolár F, Oštádal B. Molecular mechanisms of cardiac protection by adaptation to chronic hypoxia. Physiol Res. 2004;53(Suppl 1):S3–13. PubMed

Garcia-Dorado D, Rodriguez-Sinovas A, Ruiz-Meana M. Gap junction-mediated spread of cell injury and death during myocardial ischemia-reperfusion. Cardiovasc Res. 2004;61:386–401. PubMed

The IONA Study group Effect of nicorandil on coronary events in patients with stable angina: The Impact Of Nicorandil in Angina (IONA) randomised trial. Lancet. 2002;359:1269–75. Erratum in: 2002;360:806. PubMed

Oštádal B, Kolár F. Cardiac Ischemia: From Injury to Protection. London: Kluwer Academic Publishers; 1999. p. 173.

Zaugg M, Schaub MC. Signaling and cellular mechanisms in cardiac protection by ischemic and pharmacological preconditioning. J Muscle Res Cell Motil. 2003;24:219–49. PubMed

Majno G, Joris I. Apoptosis, oncosis, and necrosis. An overview of cell death. Am J Pathol. 1995;146:3–15. PubMed PMC

Haunstetter A, Izumo S. Future perspectives and potential implications of cardiac myocyte apoptosis. Cardiovasc Res. 2000;45:795–801. PubMed

Eefting F, Rensing B, Wigman J, et al. Role of apoptosis in reperfusion injury. Cardiovasc Res. 2004;61:414–26. PubMed

Becker LB. New concepts in reactive oxygen species and cardiovascular reperfusion physiology. Cardiovasc Res. 2004;61:461–70. PubMed

Piper HM, Garcia-Dorado D, Ovize M. A fresh look at reperfusion injury. Cardiovasc Res. 1998;38:291–300. PubMed

Bolli R, Marbán E. Molecular and cellular mechanisms of myocardial stunning. Physiol Rev. 1999;79:609–34. PubMed

Widimsky P, Groch L, Zelizko M, Aschermann M, Bednar F, Suryapranata H. Multicentre randomized trial comparing transport to primary angioplasty vs immediate thrombolysis vs combined strategy for patients with acute myocardial infarction presenting to a community hospital without a catheterization laboratory. The PRAGUE study. Eur Heart J. 2000;21:823–31. PubMed

Widimsky P, Budesinsky T, Vorac D, et al. ‘PRAGUE’ Study Group Investigators Long distance transport for primary angioplasty vs immediate thrombolysis in acute myocardial infarction. Final results of the randomized national multicentre trial – PRAGUE-2. Eur Heart J. 2003;24:94–104. PubMed

Oštádal P. What is reperfusion injury? Eur Heart J. 2005 (in press) PubMed

Garcia-Dorado D. Myocardial reperfusion injury: A new view. Cardiovasc Res. 2004;61:363–4. PubMed

Halestrap AP, Clarke SJ, Javadov SA. Mitochondrial permeability transition pore opening during myocardial reperfusion – a target for cardioprotection. Cardiovasc Res. 2004;61:372–85. PubMed

Hausenloy DJ, Yellon DM. New directions for protecting the heart against ischaemia-reperfusion injury: Targeting the Reperfusion Injury Salvage Kinase (RISK)-pathway. Cardiovasc Res. 2004;61:448–60. PubMed

Menasché P. Stem cells: Where we stand. Dialogues Cardiovasc Med. 2003;8:123–33.

Beltrami AP, Urbanek K, Kajstura J, et al. Evidence that human cardiac myocytes divide after myocardial infarction. N Engl J Med. 2001;344:1750–7. PubMed

Anversa P, Nadal-Ginard B. Myocyte renewal and ventricular remodelling. Nature. 2002;415:240–3. PubMed

Reffelmann T, Kloner RA. Cellular cardiomyoplasty – cardiomyocytes, skeletal myoblasts, or stem cells for regenerating myocardium and treatment of heart failure? Cardiovasc Res. 2003;58:358–68. PubMed

Oštádal B, Oštádalová I, Dhalla NS. Development of cardiac sensitivity to oxygen deficiency: Comparative and ontogenetic aspects. Physiol Rev. 1999;79:635–59. PubMed

Oštádalová I, Oštádal B, Jarkovská D, Kolár F. Ischemic preconditioning in chronically hypoxic neonatal rat heart. Pediatr Res. 2002;52:561–7. PubMed

Ranki HJ, Budas GR, Crawford RM, Davies AM, Jovanovic A. 17Beta-estradiol regulates expression of KATP channels in heart-derived H9c2 cells. J Am Coll Cardiol. 2002;40:367–74. PubMed

Bowles D. A radical idea: Men and women are different. Cardiovasc Res. 2004;61:5–6. PubMed