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Antibody formation against beta-tubulin class III in response to brain trauma
Skoda D, Kranda K, Bojar M, Glosová L, Bäurle J, Kenney J, Romportl D, Pelichovská M, Cvachovec K.
Language English Country United States
Grant support
NR8114
MZ0
CEP Register
Digital library NLK
Full text - Část
Source
NLK
ScienceDirect (archiv)
from 1993-01-01 to 2009-12-31
ROAD: Directory of Open Access Scholarly Resources
- MeSH
- Time Factors MeSH
- Financing, Organized MeSH
- Immunoglobulin G blood MeSH
- Immunoglobulin M blood MeSH
- Humans MeSH
- Brain immunology MeSH
- Neurons immunology MeSH
- Brain Injuries immunology blood MeSH
- Tubulin immunology MeSH
- Antibody Formation MeSH
- Check Tag
- Humans MeSH
Brain trauma typically leads to neuronal damage and loss. Assuming a transient autoimmune response to debris of the damaged neurones, we have monitored serum titres of IgG and IgM antibodies to beta-tubulin class III (betaTcIII), which is almost exclusively found in neuronal cytoskeletons. In 15 out of 18 patients, the peak of the IgG or IgM antibody titre appeared in the serum within 3 weeks of a brain trauma.
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- 314 __
- $a Neurology Department, 2nd Medical Faculty, Charles University, Prague, Czech Republic
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- $a Brain trauma typically leads to neuronal damage and loss. Assuming a transient autoimmune response to debris of the damaged neurones, we have monitored serum titres of IgG and IgM antibodies to beta-tubulin class III (betaTcIII), which is almost exclusively found in neuronal cytoskeletons. In 15 out of 18 patients, the peak of the IgG or IgM antibody titre appeared in the serum within 3 weeks of a brain trauma.
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