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Genetic predisposition of human plasma triglyceride concentrations
L. Schwarzova, J. A. Hubacek, M. Vrablik
Language English Country Czech Republic
Document type Journal Article, Research Support, Non-U.S. Gov't, Review
Grant support
NT12217
MZ0
CEP Register
Digital library NLK
Full text - Article
Source
NLK
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- MeSH
- Genome-Wide Association Study methods MeSH
- Genetic Predisposition to Disease genetics MeSH
- Hypertriglyceridemia blood diagnosis genetics MeSH
- Humans MeSH
- Triglycerides blood genetics MeSH
- Check Tag
- Humans MeSH
- Publication type
- Journal Article MeSH
- Research Support, Non-U.S. Gov't MeSH
- Review MeSH
The issue of plasma triglyceride levels relative to the risk of development of cardiovascular disease, as well as overall mortality, has been actively discussed for many years. Like other cardiovascular disease risk factors, final plasma TG values have environmental influences (primarily dietary habits, physical activity, and smoking), and a genetic predisposition. Rare mutations (mainly in the lipoprotein lipase and apolipoprotein C2) along with common polymorphisms (within apolipoprotein A5, glucokinase regulatory protein, apolipoprotein B, apolipo-protein E, cAMP responsive element binding protein 3-like 3, glycosylphosphatidylinositol-anchored HDL-binding protein 1) play an important role in determining plasma TG levels.
References provided by Crossref.org
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