Effect of scarlet fever toxin on the phagocytic activity of the reticuloendothelial system
Language English Country United States Media print
Document type Comparative Study, Journal Article
PubMed
393597
DOI
10.1007/bf02927125
Knihovny.cz E-resources
- MeSH
- Bacterial Toxins pharmacology MeSH
- Endotoxins pharmacology MeSH
- Escherichia coli MeSH
- Exotoxins pharmacology MeSH
- Phagocytosis drug effects MeSH
- Fever chemically induced MeSH
- Colloids MeSH
- Rabbits MeSH
- Metabolic Clearance Rate MeSH
- Mononuclear Phagocyte System drug effects physiology MeSH
- Mice MeSH
- Thorium Dioxide pharmacology MeSH
- Salmonella paratyphi B MeSH
- Scarlet Fever microbiology MeSH
- Streptococcus pyogenes analysis MeSH
- Carbon metabolism MeSH
- Animals MeSH
- Check Tag
- Rabbits MeSH
- Male MeSH
- Mice MeSH
- Female MeSH
- Animals MeSH
- Publication type
- Journal Article MeSH
- Comparative Study MeSH
- Names of Substances
- Bacterial Toxins MeSH
- Endotoxins MeSH
- Exotoxins MeSH
- Colloids MeSH
- Thorium Dioxide MeSH
- Carbon MeSH
In normal rabbits and mice, one i.v. injection of scarlet fever toxin (ET) (30 000 STD per kg of rabbit weight or 20-g mouse) elicited a similar biphasic change in carbon clearance rate - early depression followed by a stimulating phase - as has been described for Gram-negative endotoxins. Prolonged depression without a subsequent stimulation phase was obtained in mice by raising the ET dose. The reasons of the discrepancy between these findings and those of Hanna and Watson (1965b) are discussed. Pyrogenic tolerance to ET is not accompanied by accelerated carbon clearance and is not impaired by RES blockade. A possible mechanism of ET tolerance is suggested.
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