Effect of scarlet fever toxin on the phagocytic activity of the reticuloendothelial system
Jazyk angličtina Země Spojené státy americké Médium print
Typ dokumentu srovnávací studie, časopisecké články
PubMed
393597
DOI
10.1007/bf02927125
Knihovny.cz E-zdroje
- MeSH
- bakteriální toxiny farmakologie MeSH
- endotoxiny farmakologie MeSH
- Escherichia coli MeSH
- exotoxiny farmakologie MeSH
- fagocytóza účinky léků MeSH
- horečka chemicky indukované MeSH
- koloidy MeSH
- králíci MeSH
- metabolická clearance MeSH
- mononukleární fagocytární systém účinky léků fyziologie MeSH
- myši MeSH
- oxid thoričitý farmakologie MeSH
- Salmonella paratyphi B MeSH
- spála mikrobiologie MeSH
- Streptococcus pyogenes analýza MeSH
- uhlík metabolismus MeSH
- zvířata MeSH
- Check Tag
- králíci MeSH
- mužské pohlaví MeSH
- myši MeSH
- ženské pohlaví MeSH
- zvířata MeSH
- Publikační typ
- časopisecké články MeSH
- srovnávací studie MeSH
- Názvy látek
- bakteriální toxiny MeSH
- endotoxiny MeSH
- exotoxiny MeSH
- koloidy MeSH
- oxid thoričitý MeSH
- uhlík MeSH
In normal rabbits and mice, one i.v. injection of scarlet fever toxin (ET) (30 000 STD per kg of rabbit weight or 20-g mouse) elicited a similar biphasic change in carbon clearance rate - early depression followed by a stimulating phase - as has been described for Gram-negative endotoxins. Prolonged depression without a subsequent stimulation phase was obtained in mice by raising the ET dose. The reasons of the discrepancy between these findings and those of Hanna and Watson (1965b) are discussed. Pyrogenic tolerance to ET is not accompanied by accelerated carbon clearance and is not impaired by RES blockade. A possible mechanism of ET tolerance is suggested.
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