Effect of starvation and chloramphenicol on acceleration of bacterial dihexyl sulfosuccinate biotransformation
Language English Country United States Media print
Document type Journal Article
PubMed
11501413
DOI
10.1007/bf02818716
Knihovny.cz E-resources
- MeSH
- Bacterial Proteins biosynthesis MeSH
- Biotransformation drug effects MeSH
- Water Pollutants, Chemical metabolism MeSH
- Chloramphenicol pharmacology MeSH
- Comamonas drug effects metabolism MeSH
- Protein Synthesis Inhibitors pharmacology MeSH
- Kinetics MeSH
- Culture Media MeSH
- Surface-Active Agents metabolism MeSH
- Succinates metabolism MeSH
- Publication type
- Journal Article MeSH
- Names of Substances
- Bacterial Proteins MeSH
- Water Pollutants, Chemical MeSH
- Chloramphenicol MeSH
- di-n-hexyl sulfosuccinate MeSH Browser
- Protein Synthesis Inhibitors MeSH
- Culture Media MeSH
- Surface-Active Agents MeSH
- Succinates MeSH
Starvation for carbon and energy sources accelerated the biotransformation of the anion-active surfactant dihexyl sulfosuccinate (DHS) by Comamonas terrigena cells. Chloramphenicol (Cm) added at different time intervals to non-starved cells inhibited the DHS transformation. The largest difference between cells treated and non-treated by Cm was observed for a 16-h-starvation period. Protein synthesis de novo during starvation enhanced the DHS biotransformation efficiency. A partial transformation of DHS in the presence of Cm indicated the constitutive character of enzymes involved in primary DHS biodegradation.
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