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Parvovirus B19 induced lupus-like syndrome with nephritis
E. Georges, Z. Rihova, R. Cmejla, PY. Decleire, C. Langen,
Jazyk angličtina Země Anglie, Velká Británie
Typ dokumentu kazuistiky, časopisecké články
NLK
ProQuest Central
od 2011-01-01 do 2016-11-30
Health & Medicine (ProQuest)
od 2011-01-01 do 2016-11-30
- MeSH
- akutní nemoc MeSH
- autoimunitní nemoci * MeSH
- biopsie MeSH
- DNA virů analýza MeSH
- glomerulonefritida farmakoterapie imunologie virologie MeSH
- glomerulus diagnostické zobrazování MeSH
- imunosupresiva terapeutické užití MeSH
- infekce viry z čeledi Parvoviridae MeSH
- lidé MeSH
- lidský parvovirus B19 izolace a purifikace MeSH
- senioři MeSH
- syndrom MeSH
- Check Tag
- lidé MeSH
- mužské pohlaví MeSH
- senioři MeSH
- Publikační typ
- časopisecké články MeSH
- kazuistiky MeSH
We report a case of a 65-year-old man who developed an acute illness with fever, arthralgia and nephritic syndrome. Antinuclear antibodies were slightly positive and complement levels were low. Renal biopsy showed exudative diffuse proliferative endocapillary glomerulonephritis with diffuse immunoglobulin (IgG, IgA, IgM) and complement deposition (C3d, C4d, C1q) on immunofluorescence. The patient was first treated with corticosteroids and mycophenolate mofetil for suspected lupus with WHO class IV glomerulonephritis. The diagnosis was questioned and a diagnosis of parvovirus B19-associated nephritis was made based on elevation of serum IgM antibodies for parvovirus B19 and detection of parvovirus B19 DNA on renal biopsy. The immunosuppressive treatment was stopped and progressive spontaneous regression of clinical and laboratory abnormalities was observed. We conclude that human parvovirus B19 infection should be considered as a cause of lupus-like symptomatology and acute glomerulonephritis.
b Nephrology Department CSL Vivalia Arlon Belgium
Citace poskytuje Crossref.org
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- $a We report a case of a 65-year-old man who developed an acute illness with fever, arthralgia and nephritic syndrome. Antinuclear antibodies were slightly positive and complement levels were low. Renal biopsy showed exudative diffuse proliferative endocapillary glomerulonephritis with diffuse immunoglobulin (IgG, IgA, IgM) and complement deposition (C3d, C4d, C1q) on immunofluorescence. The patient was first treated with corticosteroids and mycophenolate mofetil for suspected lupus with WHO class IV glomerulonephritis. The diagnosis was questioned and a diagnosis of parvovirus B19-associated nephritis was made based on elevation of serum IgM antibodies for parvovirus B19 and detection of parvovirus B19 DNA on renal biopsy. The immunosuppressive treatment was stopped and progressive spontaneous regression of clinical and laboratory abnormalities was observed. We conclude that human parvovirus B19 infection should be considered as a cause of lupus-like symptomatology and acute glomerulonephritis.
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