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Influence of micro- and nanoplastics on mitochondrial function in the cardiovascular system: a review of the current literature
T. Lerchner, J. Jedlička, M. Kripnerová, J. Dejmek, J. Kuncová
Status minimal Language English Country Czech Republic
Document type Journal Article, Review
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PubMed Central
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from 2005-01-01
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- MeSH
- Cardiovascular Diseases metabolism MeSH
- Cardiovascular System * metabolism drug effects MeSH
- Humans MeSH
- Microplastics toxicity MeSH
- Mitochondria * metabolism drug effects MeSH
- Nanoparticles MeSH
- Reactive Oxygen Species metabolism MeSH
- Animals MeSH
- Check Tag
- Humans MeSH
- Animals MeSH
- Publication type
- Journal Article MeSH
- Review MeSH
Mitochondria represent pivotal cellular organelles endowed with multifaceted functionalities encompassing cellular respiration, metabolic processes, calcium turnover, and the regulation of apoptosis, primarily through the generation of reactive oxygen species (ROS). Perturbations in mitochondrial dynamics have been intricately linked to the etiology of numerous cardiovascular pathologies, such as heart failure, ischemic heart disease, and various cardiomyopathies. Notably, recent attention has been directed towards the detrimental impact of micro- and nanoplastic pollution on mitochondrial integrity, an area underscored by a paucity of comprehensive investigations. Given the escalating prevalence of plastic particle contamination and the concomitant burden of cardiovascular disease in aging populations, understanding the interplay between mitochondria within the cardiovascular system and micro- and nanoplastic pollution assumes paramount importance. This review endeavors to elucidate the current albeit limited comprehension surrounding this complex interplay. Key words Mitochondria, Nanoplastics, Microplastics, Cardiovascular system, Endothelial function, Oxidative phosphorylation.
References provided by Crossref.org
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