Karyotype evolution and acquisition of FLT3 or RAS pathway alterations drive progression of myelodysplastic syndrome to acute myeloid leukemia
Language English Country Italy Media print-electronic
Document type Letter
PubMed
26294738
PubMed Central
PMC4666337
DOI
10.3324/haematol.2015.127985
PII: haematol.2015.127985
Knihovny.cz E-resources
- Keywords
- MDS, mutation analysis, s-AML,
- MeSH
- Abnormal Karyotype * MeSH
- Leukemia, Myeloid, Acute genetics pathology MeSH
- Middle Aged MeSH
- Humans MeSH
- Myelodysplastic Syndromes genetics pathology MeSH
- ras Proteins genetics MeSH
- Aged, 80 and over MeSH
- Aged MeSH
- fms-Like Tyrosine Kinase 3 genetics MeSH
- Check Tag
- Middle Aged MeSH
- Humans MeSH
- Male MeSH
- Aged, 80 and over MeSH
- Aged MeSH
- Female MeSH
- Publication type
- Letter MeSH
- Names of Substances
- FLT3 protein, human MeSH Browser
- ras Proteins MeSH
- fms-Like Tyrosine Kinase 3 MeSH
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