Clinical impact of low-burden BCR-ABL1 mutations detectable by amplicon deep sequencing in Philadelphia-positive acute lymphoblastic leukemia patients
Language English Country England, Great Britain Media print-electronic
Document type Letter, Research Support, Non-U.S. Gov't
PubMed
26867670
DOI
10.1038/leu.2016.17
PII: leu201617
Knihovny.cz E-resources
- MeSH
- Precursor Cell Lymphoblastic Leukemia-Lymphoma diagnosis drug therapy genetics MeSH
- Fusion Proteins, bcr-abl antagonists & inhibitors genetics MeSH
- Drug Resistance, Neoplasm genetics MeSH
- Adult MeSH
- Protein Kinase Inhibitors therapeutic use MeSH
- Middle Aged MeSH
- Humans MeSH
- Neoplasm Recurrence, Local diagnosis genetics MeSH
- Mutation MeSH
- DNA Mutational Analysis methods MeSH
- Antineoplastic Agents therapeutic use MeSH
- Neoplasm, Residual diagnosis genetics MeSH
- Aged MeSH
- High-Throughput Nucleotide Sequencing methods MeSH
- Check Tag
- Adult MeSH
- Middle Aged MeSH
- Humans MeSH
- Male MeSH
- Aged MeSH
- Female MeSH
- Publication type
- Letter MeSH
- Research Support, Non-U.S. Gov't MeSH
- Names of Substances
- Fusion Proteins, bcr-abl MeSH
- BCR-ABL1 fusion protein, human MeSH Browser
- Protein Kinase Inhibitors MeSH
- Antineoplastic Agents MeSH
Bone Marrow Transplant Unit Department of Oncology and Hematology University of Modena Modena Italy
Hematology Department of Cellular Biotechnologies and Hematology La Sapienza University Rome Italy
Hematology S Eugenio Hospital Tor Vergata University Rome Italy
Hematology Università Cattolica del Sacro Cuore Rome Italy
Institute of Hematology and Blood Transfusion Prague Czech Republic
MLL Munich Leukemia Laboratory Munich Germany
Onco Hematology Division IRCCS Ca' Granda Maggiore Policlinico Hospital Foundation Milan Italy
Personalised Healthcare and Biomarkers AstraZeneca Cambridge UK
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