Eight novel loci implicate shared genetic etiology in multiple myeloma, AL amyloidosis, and monoclonal gammopathy of unknown significance
Language English Country England, Great Britain Media print-electronic
Document type Letter, Research Support, Non-U.S. Gov't
PubMed
31695157
DOI
10.1038/s41375-019-0619-1
PII: 10.1038/s41375-019-0619-1
Knihovny.cz E-resources
- MeSH
- Genome-Wide Association Study methods MeSH
- Genetic Loci * MeSH
- Polymorphism, Single Nucleotide * MeSH
- Cohort Studies MeSH
- Humans MeSH
- Multiple Myeloma etiology pathology MeSH
- Monoclonal Gammopathy of Undetermined Significance etiology pathology MeSH
- Biomarkers, Tumor genetics MeSH
- Immunoglobulin Light-chain Amyloidosis etiology pathology MeSH
- Prognosis MeSH
- Check Tag
- Humans MeSH
- Publication type
- Letter MeSH
- Research Support, Non-U.S. Gov't MeSH
- Names of Substances
- Biomarkers, Tumor MeSH
Department of Biology University of Pisa Pisa Italy
Department of Biomedicine University of Basel Basel Switzerland
Department of Genomics Life and Brain Research Center University of Bonn Bonn Germany
Department of Internal Medicine 5 University of Heidelberg Heidelberg Germany
Department of Medical Biosciences Pathology University of Umea Umea Sweden
Division of Pediatric Neurooncology German Cancer Research Center Heidelberg Germany
Hopp Children's Cancer Center Heidelberg Germany
See more in PubMed
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Haplotype analysis identifies functional elements in monoclonal gammopathy of unknown significance
Epidemiology, genetics and treatment of multiple myeloma and precursor diseases
Search for AL amyloidosis risk factors using Mendelian randomization
Search for multiple myeloma risk factors using Mendelian randomization
