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A metabolic switch in proteasome inhibitor-resistant multiple myeloma ensures higher mitochondrial metabolism, protein folding and sphingomyelin synthesis
L. Besse, A. Besse, M. Mendez-Lopez, K. Vasickova, M. Sedlackova, P. Vanhara, M. Kraus, J. Bader, RB. Ferreira, RK. Castellano, BK. Law, C. Driessen,
Language English Country Italy
Document type Letter, Research Support, Non-U.S. Gov't
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- MeSH
- Bortezomib pharmacology MeSH
- Drug Resistance, Neoplasm * MeSH
- Homeostasis MeSH
- Proteasome Inhibitors pharmacology MeSH
- Middle Aged MeSH
- Humans MeSH
- Lipids chemistry MeSH
- Metabolomics MeSH
- Mitochondria metabolism MeSH
- Multiple Myeloma drug therapy MeSH
- Cell Line, Tumor MeSH
- Oligopeptides pharmacology MeSH
- Hydrogen Peroxide chemistry MeSH
- Proteasome Endopeptidase Complex metabolism MeSH
- Antineoplastic Agents pharmacology MeSH
- Antineoplastic Combined Chemotherapy Protocols pharmacology MeSH
- Reactive Oxygen Species metabolism MeSH
- Gene Expression Regulation, Neoplastic MeSH
- Protein Folding MeSH
- Aged MeSH
- Sphingomyelins biosynthesis MeSH
- Gene Expression Profiling MeSH
- Check Tag
- Middle Aged MeSH
- Humans MeSH
- Aged MeSH
- Female MeSH
- Publication type
- Letter MeSH
- Research Support, Non-U.S. Gov't MeSH
Department of Chemistry University of Florida Gainesville FL USA
Department of Histology and Embryology Faculty of Medicine Masaryk University Brno Czech Republic
References provided by Crossref.org
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